P2Y-like receptor, GPR105 (P2Y (sub)14), identifies and mediates chemotaxis of bone-marrow hematopoietic stem cells

Article Abstract:

Research has been conducted on P2Y receptors which are G-protein-coupled nucleotide receptor responsible for immune responses to injury. The authors report the identification of P2Y-like receptor for UDP-conjugated sugars GPR105 (P2Y (sub)14) with restricted primitive cell expression in the hematopoietic lineage.

Pharmaceutical Preparation Manufacturing, Pharmaceutical preparations, Biological Product (except Diagnostic) Manufacturing, Carbohydrates, Nucleic Acid Derivatives, Causes of, Bone marrow, Chemotaxis, G proteins, Stem cells, Hematopoiesis, Nucleotides

---

Definition of a novel growth factor-dependent signal cascade for the suppression of bile acid biosynthesis

Article Abstract:

Research has been conducted on nuclear bile acid receptor. The authors have tested the hypotheses that this receptor is important for the gene encoding cholesterol 7(alpha)-hydroxylase which is the first step in the bile acid biosynthesis, and they report the results.

Growth factors, Biosynthesis, Cholesterol, Bile acids

---

Similar abstracts:

• Abstracts: 16S rRNA-based probes and polymerase chain reaction method to detect Listeria monocytogenes cells added to foods
• Abstracts: Composition, variation, and dynamics of major osmotic solutes in Methanohalophilus strain FDF1. Halotolerance in Methanosarcina spp.: role of N(super epsilon)-acetyl-beta-lysine, alpha-glutamate, glycine betaine, and K+ as compatible solutes for osmotic adaptation
• Abstracts: Specific recruitment of SH-PTP1 to the erythropoietin receptor causes inactivation of JAK2 and termination of proliferative signals
• Abstracts: TRF1 is degraded by ubiquitin-mediated proteolysis after release from telomeres. How does calorie restriction work?
• Abstracts: Photoreceptor ubiquitination by COP1 E3 ligase desensitizes phytochrome A signaling. The POU proteins Brn-2 and Oct-6 share important functions in Schwann cell development

This website is not affiliated with document authors or copyright owners. This page is provided for informational purposes only. Unintentional errors are possible.