Cytotoxicity associated with trichloroethylene oxidation in Burkholderia cepacia G4

Article Abstract:

Trichloroethylene (TCE) oxidation by Burkholderia cepacia G4 is toxic to the bacterium. This occurs because reactive intermediates generated during TCE oxidation can inactivate not only the oxygenase, but also other cellular constituents as well.

Health aspects

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Requirement of DNA repair mechanisms for survival of Burkholderia cepacia G4 upon degradation of trichloroethylene

Article Abstract:

Burkholderia cepacia G4 can degrade trichloroethylene (TCE) as long as its DNA repair mechanism is intact. This bacterium is more resistant to the toxic effects of TCE than other bacteria and for this reason is considered a potential agent for TCE biodegradation.

DNA repair

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Inactivation of toluene 2-monooxygenase in Burkholderia cepacia G4 by alkynes

Article Abstract:

Research was conducted to examine the inactivation of toluene 2-monooxygenase in Burkholderia cepacia G4 by alkynes. Results demonstrate that different alkynes serve as potent inhibitors of the toluene-dependent growth of B. cepacia. Findings also reveal that toluene 2-monooxygenase activity is specifically inactivated by the compounds. Therefore, alkynes represent a general class of mechanism-based inactivators for toluene 2-monooxygenase activity in B. cepacia G4.

Bacterial growth, Oxidases, Toluene, Acetylene

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