Notch signaling regulates left-right asymmetry determination by inducing Nodal expression

Article Abstract:

Research has been conducted on left-right asymmetry generation in the vertebrate body. The authors demonstrate that Notch signaling is important for the asymmetry establishment in mice carrying it out via Nodal gene expression regulation.

Japan, Analysis, Physiological aspects, Genetic regulation, Gene expression, Genetic research

---

Roles for Nkx3.1 (ital) in prostate development and cancer

Article Abstract:

The mouse Nkx3.1 (ital) homeobox gene has roles in prostate development and in cancer. This gene is the earliest known marker of prostate epithelium in embryogenesis and is expressed after that at all stages of prostate differentiation in vivo and in tissue recombinants. NKX3.1 (ital) may be a prostate-specific tumor suppressor gene. Loss a single allele may constitute a predisposition to carcinogenesis in the prostate.

Research, Usage, Observations, Cancer, Mice, mutant strains, Mutant mice, Cancer genetics, Cell differentiation, Biological models, Homeobox genes, Tumor suppressor genes, Dysplasia, Prostate, Hyperplasia, Bulbourethral glands

---

Similar abstracts:

• Abstracts: Neurogenin promotes neurogenesis and inhibits glial differentiation by independent mechanisms. Orchestral maneuvers in the axon: trio and the control of axon guidance
• Abstracts: Lateral signaling mediated by axon contact and calcium entry regulates asymmetric odorant receptor expression in C. elegans
• Abstracts: Functionally related motor neuron pool and muscle sensory afferent subtypes defined by coordinate ETS gene expression
• Abstracts: Horizontal heterogeneity of denitrifying bacterial communities in marine sediments by terminal restriction fragment length polymorphism analysis
• Abstracts: Monitoring precursor 16S rRNAs of Acinetobacter spp. in activated sludge waste-water treatment systems. Monitoring precursor 16S rRNAs of Acinetobacter spp. in activated sludge wastewater treatment systems

This website is not affiliated with document authors or copyright owners. This page is provided for informational purposes only. Unintentional errors are possible.