Protection of acid-base balance by pH regulation of acid production

Article Abstract:

The body can compensate for increased production of ketoacids and lactic acid by adjusting the rate of their formation. Normally, the blood is maintained at a neutral pH because the kidneys excrete hydrogen ions generated during metabolic processes. However, if carbohydrates or fats are not completely oxidized, the body produces ketoacids and lactic acid. If produced in large quantities, they can cause excess acid in the body, a condition called acidosis. Studies have shown that the excess acid causes blood pH to drop, which inhibits the production of ketoacids.

Prevention, Physiological aspects, Hydrogen-ion concentration, Acid-base imbalances, Acid-base imbalance, Ketoacidosis, Diabetic ketoacidosis

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Arachidonate 5-lipoxygenase promoter genotype, dietary arachidonic acid, and atherosclerosis

Article Abstract:

Two particular mutations in the gene for the enzyme 5-lipoxygenase may increase the risk of atherosclerosis, according to a study of 470 people. Those who had one of the two mutations were more likely to have atherosclerosis in the carotid arteries, which are in the neck. Dietary n-6 polyunsaturated fatty acids promoted carotid artery atherosclerosis in these people, whereas marine n-3 fatty acids inhibited atherosclerosis.

Atherosclerosis

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The genotype of the original Wiskott phenotype

Article Abstract:

A new frame shift mutation in exon 1 of Wiskott-Aldrich syndrome (WAS) is identified to investigate family members of the patients originally described by Wiskott in 1937. It is reported that the mutation is likely to be the hypothesized genotype that causes the severe form of WAS.

Care and treatment, Diagnosis, Wiskott-Aldrich syndrome, Disease/Disorder overview

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