Hyperamylasemia in bulimia nervosa and hyperemesis gravidarum

Article Abstract:

There is a raised total serum amylase but no rise in pancreatic amylase in a large proportion of hyperemetic patients, according to research undertaken among pregnant women with hyperemesis gravidarum, nonvomiting pregnant women and female bulimic patients. This finding suggests that the source of the increased amylase is the salivary glands. It appears that the raised amylase in bulimia nervosa and hyperemsis is caused by vomiting. In bulimia nervosa, vomiting repeatedly stimulates the salivary glands, and this may lead to increased amylase production and enlargement of salivary glands.

Hyperemesis gravidarum

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Brain hypometabolism of glucose in bulimia nervosa

Article Abstract:

Bulimia nervosa is a condition characterized by recurrent episodes of binge eating and compensatory behaviors, such as purging or fasting, to prevent weight gain. To investigate brain glucose metabolism in patients with bulimia nervosa, positron emission tomography with (18-F)-fluorodeoxyglucose was used. Results showed that bulimic patients exhibited global and regional absolute hypometabolism of glucose. Such an observation could be a consequence of neurobiological perturbations following nutritional deficiencies or a particular cerebral dysfunction in eating disorders.

Psychological aspects, Brain research, Glucose metabolism

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Electrolyte and other blood serum abnormalities in normal weight bulimia nervosa: Evidence for sampling bias

Article Abstract:

The prevalence of electrolyte and other serum abnormalities in bulimia nervosa patients was investigated. None of the subjects displayed electrolyte abnormalities, but hypomagnesemia and hypoalbuminemia were found, although the severity of the abnormalities was modest.

Electrolyte metabolism

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