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Evidence that human cardiac myocytes divide after myocardial infarction

Article Abstract:

Researchers show that heart muscle can repair itself to an extent after a heart attack. They found that heart muscle cells around the area that was damaged begin dividing to a greater extent than cells in areas that were not damaged.

Author: Anversa, Piero, Nadal-Ginard, Bernardo, Beltrami, Antonio P., Urbanek, Konrad, Kajstura, Jan, Yan, Shao-Min, Finato, Nicoleetta, Bussani, Rossana, Silvestri, Furio, Leri, Annarosa, Beltrami, C. Alberto
Publisher: Massachusetts Medical Society
Publication Name: The New England Journal of Medicine
Subject: Health
ISSN: 0028-4793
Year: 2001
Heart attack, Mitosis

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Pim-1 regulates cardiomyocyte survival downstream of Akt

Article Abstract:

The regulation of cellular proliferation and survival by the serine-threonine kinases Pim-1 and Akt is discussed. Some studies show that Pim-1 plays a major role in cardioprotection downstream of Akt.

Author: Berns, Anton, Anversa, Piero, Kajstura, Jan, Leri, Annarosa, Rota, Marcello, Houser, Steven R., Muraski, John A., Yu Misao, Fransioli, Jenna, Cottage, Christopher, Gude, Natalie, Esposito, Grazia, Delucchi, Francesca, Arcarese, Michael, Alvarez, Roberto, Siddiqi, Sailay, Emmanuel, Gregory N., Wu, Weitao, Fischer, Kimberlee, Martindale, Joshua J., Glembotski, Christopher C., Magnuson, Nancy, Beretta, Remus M., Schaefer, Erik M., Sussman, Mark A.
Publisher: Nature America, Inc.
Publication Name: Nature Medicine
Subject: Health
ISSN: 1078-8956
Year: 2007
Science & research, Genetic aspects, Gene expression, Serine, Threonine, Chemical properties, Phosphotransferases

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Apoptosis in the failing human heart

Article Abstract:

A process of natural cell death called apoptosis appears to occur in heart cells during heart failure. Researchers used microscopy and various chemical methods to study apoptosis in the hearts of 36 patients with heart failure who were receiving a heart transplant and 3 patients who died of heart attacks. Apoptosis of heart cells increased over 200 times the normal rate seen in hearts from 11 people who died of other causes. This was true even though the cells were expressing a protein that protects against apoptosis.

Author: Anversa, Piero, Reed, John C., Kajstura, Jan, Beltrami, Carlo A., Cheng, Wei, Krajewski, Stanislaw, Olivetti, Giorgio, Abbi, Rakesh, Quaini, Federico, Nitahara, James A., Quaini, Eugenio, Di Loreto, Carla
Publisher: Massachusetts Medical Society
Publication Name: The New England Journal of Medicine
Subject: Health
ISSN: 0028-4793
Year: 1997
Analysis, Cell death, Muscle cells, Heart failure

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Subjects list: Research, Physiological aspects, Heart muscle, Myocardium
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