Growth on urea can trigger death and peroxidation of the cyanobacterium Synechococcus sp. strain PCC 7002

Article Abstract:

Studies were conducted to investigate the possible cause of rapid-death of urease producing cyanobacteria in natural blooms. The oxidation of pigmentation and the ability of vitamin E to delay the onset of cell death suggest that peroxidation of the lipids occur in the cell death phase. Moreover, rapid cell death may have occurred as a result of the increase in intracellular pH after the decomposition of carbonic acid and ammonia resulting from the hydrolysis of urea.

Physiological aspects, Urea

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Potassium salts inhibit growth of the cyanobacteria Microcystis spp. in pond water and defined media: implications for control of microcystin-producing aquatic blooms

Article Abstract:

Ten metals were assayed in 21 Indian ponds classified into three groups. These groups are: eutrophic alkaline ponds with < 2.5 mM potassium and thick growths of Microcystis aeruginosa or Microcystis flos-aquae during most of the year; equally eutrophic alkaline ponds with > 2.8 mM potassium and no discernible Microcystis growth; and oligo- or mesotrophic ponds with various potassium and hydrogen ion concentrations and no persistent Microcystis blooms.

Potassium (Chemical element), Potassium

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Bactericidal activity of photocatalytic TiO2 reaction: toward an understanding of its killing mechanism

Article Abstract:

Research was conducted to prove for the first time that the TiO2 photocatalytic reaction triggers the lipid peroxidation reaction which results in the normal functions related to an intact membrane such as respiratory activity to be lost. It is proposed that the loss of membrane structure and, subsequently, membrane functions is the root cause of cell death when photocatalytic TiO2 particles are outside the cell.

Antibacterial agents, Titanium dioxide, Lipid peroxidation

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