Calmodulin kinase II inhibition protects against structural heart disease

Article Abstract:

An attempt is made to show that calmodulin-dependent protein kinase II (CaMKII) inhibition substantially prevented maladaptive remodeling from excessive beta-AR stimulation and myocardial infarction. The finding mark CaMKII as a determinant of clinically important heart disease phenotypes, and suggest CaMKII inhibition can be a highly selective approach for targeting adverse myocardial remodeling linked to betaAR signaling.

Prevention, Genetic aspects, Heart diseases, Calmodulin

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Inhibition of CDK1 as a potential therapy for tumors over-expressing MYC

Article Abstract:

The authors propose through their presentation that CDK1 inhibition may give positive results in the treatment of human malignancies overexpressing MYC.

Care and treatment, Cell cycle, Tumors, Cell research, Cytological research, Tumours, Properties

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