Prohibitions control cell proliferation and apoptosis by regulating OPA1-dependent cristae morphogenesis in mitochondria

Article Abstract:

Several conditional gene targeting studies are conducted to explain the different cellular activities of prohibitions that consist of a conserved family of membrane proteins. The prohibitions are found to effectively control the cell proliferation and apoptosis by regulating the OPA1-dependent cristae morphogenesis in mitochondria.

Genetic aspects, Observations, Membrane proteins, Fluorescence microscopy, Mitochondria

---

SIRT1 controls endothelial angiogenic functions during vascular growth

Article Abstract:

A study examined the role of SIRT1 during vascular blood vessel growth and its control on the angiogenic activity of endothelial cells. The findings revealed that SIRT1 acts as a modulator of endothelial gene expression governing postnatal vascular growth.

Analysis, Gene expression, Vascular endothelium, Glycocalyx

---

Modulation of mammalian life span by the short isoform of p53

Article Abstract:

Findings reveal that pathways of gene activity are linked in mammals via p53 to mechanisms controlling cell proliferation. The importance of appropriate expression of the short and long p53 isoforms in lengthening the mammalian life spans is highlighted.

Evaluation, Mammals, Life spans (Biology), Life span (Biology)

Similar abstracts:

• Abstracts: DNA extraction and PCR methods for the detection of Listeria monocytogenes in cold-smoked salmon. Development of a PCR-enzyme immunoassay oligoprobe deection method for Toxoplasma gondii oocysts, incorporating PCR controls
• Abstracts: Tissue fusion and cell sorting in embryonic development and disease: Biomedical implications. The cadherin-catenin complex as a focal point of cell adhesion and signalling: new insights from three-dimensional structures
• Abstracts: Aurora-A acts as a tumor suppressor and regulates self-renewal of Drosophila neuroblasts. OVCAI: emerging as a bona fide tumor suppressor
• Abstracts: CHOP is implicated in programmed cell death in response to impaired function of the endoplasmic reticulum. p53 and stress in the ER.

This website is not affiliated with document authors or copyright owners. This page is provided for informational purposes only. Unintentional errors are possible.