Nitric oxide as a mediator of hemodynamic abnormalities and sodium and water retention in cirrhosis

Article Abstract:

Animal research indicates that nitric oxide may be involved in the development of ascites in patients with cirrhosis. Cirrhosis is a liver disease that can lead to an abnormal accumulation of fluid in body tissues, a condition called ascites. Nitric oxide dilates blood vessels and many of the blood flow abnormalities seen in cirrhosis can be ascribed to widespread dilation of arteries. When rats with cirrhosis are given a chemical that inhibits the production of nitric oxide, excess sodium and water are excreted. Humans with cirrhosis have high nitric oxide levels but its role in ascites is not yet clear.

Nitric oxide, Ascites, Liver cirrhosis

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Hormones and hemodynamics in heart failure

Article Abstract:

Research has shed much light on the cause of heart failure. As the heart begins to fail, it cannot pump as much blood as before. There are cells inside arteries that can detect low blood flow through the artery. These cells trigger several mechanisms to maintain adequate blood flow to other organs. However, these mechanisms may have a negative side, causing fluid to accumulate in various tissues, as well as other effects. For this reason, doctors often use ACE inhibitors and beta blockers in addition to diuretics to treat heart failure.

Heart failure

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Acute renal failure and sepsis

Article Abstract:

Various studies show that sepsis is associated with high incidence of acute renal failure. Persons with acute renal failure who do not have sepsis have lower mortality than those with sepsis related acute renal failure.

Science & research, Research, Mortality, Causes of, Risk factors, Influence, Bacterial infections, Acute renal failure, Acute kidney failure, Sepsis

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