SAP-less chromatin triggers systemic lupus erythematosus

Article Abstract:

Serum amyloid protein (SAP)-less chromatin has been found to trigger systemic lupus erythematosus (SLE). SAP is highly conserved in vertebrates and invertebrates and appears to give protection against autoimmune disease. Questions about SLE exist, among them whether the suspect nuclear antigens do initiate the autoimmune response. The elicited autoantibodies may or may not themselves be pathogenic. The immune mediators that allow or promote unchecked autoreactivity are yet to be identified.

Physiological aspects, Autoimmune diseases, Chromatin, Systemic lupus erythematosus

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Building global networks for human diseases: genes and populations

Article Abstract:

The forum on "Building Global Networks for Human Diseases: Genes and Populations" is concerned with the genetic study of human disease. The forum will need biobanks to play a vital role in the identification of genes associated with disease and the collaborations at a global scale would maximize their impact and chances of success.

Science & research, Evaluation, Conferences, meetings and seminars, Genetic research

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